By David C. Preston, Barbara E. Shapiro
Diagnose neuromuscular problems extra speedy and appropriately with Electromyography and Neuromuscular issues: Clinical-Electrophysiologic Correlations, third variation! state of the art suggestions is helping you correlate electromyographic and medical findings and use the most recent EMG innovations to their fullest potential.
Successfully correlate electrodiagnostic findings with key medical findings for extra convinced diagnoses.
Clearly see the way to observe what you've realized with considerable case reports in the course of the book.
Obtain suitable scientific information fast and simply with an available, easy-to-read writing type that's either complete and straightforward to understand.
Ensure right EMG needle placement and keep away from neurovascular accidents via touching on greater than sixty five special, cross-sectional anatomy drawings.
Diagnose many newly outlined genetic neuromuscular stipulations according to their electrodiagnostic presentation.
Stay brand new with must-know details on iatrogenic issues of electrodiagnostic studies.
Visualize key thoughts extra simply with a brand-new full-color layout, new paintings, and new pictures.
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Extra resources for Electromyography and Neuromuscular Disorders: Clinical-Electrophysiologic Correlations (3rd Edition)
Conduction velocities usually are measured in meters per second (m/s). It is essential to note that both latency and conduction velocity reflect only the fastest conducting fibers in the nerve being studied. By definition, conduction along these fibers arrives first and thus it is these fibers that are the ones measured. The many other slower conducting fibers participate in the CMAP area and amplitude but are not 21 22 SECTION II Fundamentals of Nerve Conduction Studies reflected in either the latency or conduction velocity measurements.
In contrast, sensory or mixed nerve conduction abnormalities always imply a primary disorder of the peripheral nerve. Neuropathic Lesions Neuropathic lesions can be divided into those that primarily affect either the axon or the myelin sheath. Axonal loss may be seen after physical disruption of the nerve or as a result of numerous toxic, metabolic, or genetic conditions that can damage the metabolic machinery of the axon. Demyelination resulting from loss or dysfunction of the myelin sheath is seen most often in entrapment or compressive neuropathies.
However, problems occur if the two potentials have a similar latency or, more importantly, if the sensory potential is absent. When the latter occurs, one can mistake the first component of the volume conducted motor potential for the SNAP, where none truly exists. It is in this situation that measuring the duration of the potential can be helpful in distinguishing a sensory from a motor potential. If one is still not sure, performing an orthodromic study will settle the issue, as no volume conducted motor response will occur with an orthodromic study.